Advisor: Charlene McQueen
My dissertation/thesis focused on hydrazine hepatotoxicity, a common manifestation of isoniazed therapy. A pathological hallmark of hydrazine hepatotoxicity in rodents is fatty liver (steatosis). The project has been successful in clarifying mechanisms involved in hepatic lipid accumulation including the involvement of PPARalpha, a novel mechanism.
I currently work as a research associate on Alzheimer's disease projects.
My dissertation/thesis focused on hydrazine hepatotoxicity, a common manifestation of isoniazed therapy. A pathological hallmark of hydrazine hepatotoxicity in rodents is fatty liver (steatosis). The project has been successful in clarifying mechanisms involved in hepatic lipid accumulation including the involvement of PPARalpha, a novel mechanism.
I currently work as a research associate on Alzheimer's disease projects.
Degree:
PhD
Program:
Pharmacology and Toxicology
Track:
Pharmacology and Toxicology
Employer:
Research Associate, School of Pharmacy, University of Wisconsin, Madison
Dissertation Title:
“N-Acetylation as Protactive against Hydrazine Hepatoxicity”
Career Type:
Academia