Joseph Herndon, PhD

Involved in laboratory research investigating various neurobiological phenomena. Currently using an in vivo model of peripheral inflammatory pain to investigate the functional upregulation of P-glycoprotein at the blood brain barrier in pain states. Previously determined mechanisms contributing to MDMA-induced neurotoxicity. Interests include neuroscience research techniques, neuroanatomy/neurophysiology, neurotherapeutics, and neurotoxicology.
Pharmacology and Toxicology
Pharmacology and Toxicology
Postdoctoral Research Associate, The University of Arizona
Dissertation Title: 
“Methylenedioxymethamphetamine-Induced Neurotoxicity: The Role of Hepatic Enzymes Cytochrome P450 2D6 and Catechol-O-Methyltransferase and Contribution of Microglia”
Honors and Achievements: 
1. NIEHS Training Grant-Environmental Toxicology of Human Diseases, 2010, 2011 (30, 31).
2. Mountain West Society of Toxicology Student Travel Award, 2011.
3. Graduate Track Award: Academic Excellence, 2012-13.
1. Herndon, J.M., Cholanians, A.B, Lau, S.S., and Monks, T.J. Glial cell response to 3,4-(±)-methylenedioxymethamphetamine and its metabolites. Toxicol Sci, 138:130-138, 2014.
2. Herndon, J.M.,Cholanians, A.B, Lau, S.S., and Monks, T.J. Catechol O-methyltransferase and 3,4-(±)methylenedioxy-methamphetamine toxicity. Toxicol. Sci, 139(1):162-73, 2014.
Career Type: